FIDRISERTIB | FIDRISERTIB : Inhibitor of ACVR1
RATINGS:
Cellular Use: (1 reviews)

In Model Organisms: (1 reviews)
Control Compounds

Probe Summary

Targets Biochemical/Biophysical Potency Cellular Potency
ACVR1 (Mutant:WT, R206H)
  • IC50:0.2 nM
  • IC50:0.6 nM
  • Kd:1.2 nM
  • Kd:2.1 nM
  • IC50:7 nM
  • IC50:150 nM
  • IC50:30 nM
Inhibitor
up to 100 nM

Selectivity

In Vitro Selectivity Assessment
Potency Assay Off-Target:
Kinome profiling of BLU-782 showed high selectivity, with only four kinases exhibiting <100 nM prote ...
In Cell Selectivity Assessment
Potency Assay Off-Target:
Cellular IC50 values showed >100-fold selectivity of BLU-782 for ALK2-R206H over ALK1, ALK3, and ALK ...

Potency
Cellular
In Vitro

ACVR1 (Mutant:WT, R206H)

Mode of Action: Inhibitor

Structure-Activity-Relationship data available? Yes

DOI Reference: 10.1126/scitranslmed.abp8334

In Vivo Validations

Mouse
Dose: 10, 25, or 50 mg/kg
Route of delivery: Oral
Target engagement assay: Measurements of SMAD1-P in tumor homogenates revealed potent inhibition of ALK2R206H signaling by BLU-782 in a time-, dose-, and exposure-dependent manner with >70% SMAD1-P inhibition for 24 hours at the 50 mg/kg oral dose (IC70 = 13.2 ng/ml in mice).

DOI Reference: 10.1126/scitranslmed.abp8334

Chemical Information

Molecular Formula C31H42N6O4
SMILEs CCOC1(c2ccc(-c3cc4c(N5CCN(C(=O)O[C@@H]6CCOC6)CC5)ccnn4c3)nc2)CCN(C(C)C)CC1
InChI InChI=1S/C31H42N6O4/c1-4-40-31(9-12-34(13-10-31)23(2)3)25-5-6-27(32-20-25)24-19-29-28(7-11-33-37(29)21-24)35-14-16-36(17-15-35)30(38)41-26-8-18-39-22-26/h5-7,11,19-21,23,26H,4,8-10,12-18,22H2,1-3H3/t26-/m1/s1
Molecular weight 562.33 Da
AlogP 0.0
HBond acceptors 10
HBond donors --
Atoms 83

References

Cross References

canSARChEMBL

Vendors

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Expert Reviews


(on 17 Feb 2026 )
Cellular Use Rating
In Model Organisms
Selectivity of Fidrisertib (Blu-782, IPN60130) has been determined by KinomeScan and in cells using constitutively active ALK1-6 receptor kinases, but data is lacking for BMP vs activin vs TGF-beta ligand-induced...
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